Atlas of Sellar and Parasellar Lesions: Clinical, by Gabriel Zada, M. Beatriz S. Lopes, Srinivasan Mukundan Jr.,

By Gabriel Zada, M. Beatriz S. Lopes, Srinivasan Mukundan Jr., Edward R. Laws Jr.

This ebook offers, in a stepwise and interactive style, nearly seventy five circumstances that mirror the huge spectrum of pathology encountered during this zone. every one case description commences with a concise medical state of affairs. top of the range radiologic, laboratory, and histopathologic photographs depicting the differentiating positive factors of the lesion subtype in query are then provided, and key operative and medical administration pearls are in short reviewed. The interdisciplinary nature of this easy-to-use colour atlas and textbook displays the truth that the administration of sufferers with sellar and parasellar lesions is itself usually interdisciplinary. The layout is exclusive in that no related interdisciplinary ebook is out there on lesions of this quarter of the mind.

Atlas of Sellar and Parasellar Lesions: scientific, Imaging, and Pathologic Correlations is of serious worth for practitioners and trainees in various scientific specialties, together with radiology, neurology, endocriniology, pathology, oncology, radiation oncology, and neurosurgery.

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Shortly before delivery, maternal oxytocin 30 M. Padilla and J. Carmichael crossing the placenta reaches the fetal brain and induces a switch in the action of GABA from excitatory to inhibitory fetal neurons [60]. This action increases the resistance of fetal neurons to hypoxia insults during labor [60]. Following parturition, oxytocin becomes an essential tool for the development of mother-child attachment [60]. Suckling stimulates a bolus release of oxytocin, which (along with prolactin) causes ejection of the milk from the mammary glands [60].

Skull Base. 2008;18:9–15. 16. Sirikci A, Bayazit YA, Bayram M, Mumbuç S, Güngör K, Kanlikama M. Variations of sphenoid and related structures. Eur Radiol. 2000;10:844–8. 17. Naganuma H, Satoh E, Nukui H. Technical considerations of transsphenoidal removal of fibrous pituitary adenomas and evaluation of collagen content and subtype in the adenomas. Neurol Med Chir. 2002;42:202–12; discussion 213. 18. Pierallini A, Caramia F, Falcone C, Tinelli E, Paonessa A, Ciddio AB, et al. Pituitary macroadenomas: preoperative evaluation of consistency with diffusion-weighted MR imaging–initial experience.

Deficiencies can be either congenital or acquired and can vary in severity from mild to complete gonadal dysfunction. Central congenital causes include a variety of genetic mutations involved in migration or function of GnRH neurons and the development and function of the gonadotroph cells. Functional causes are numerous; they include excessive exercise, stress, malnutrition, obesity, chronic illness, depression, and medications including opiates and antidepressants. Tumors, infiltrative diseases of the pituitary and hypothalamus, trauma, and malignancies also can lead to hypogonadotropic hypogonadism.

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