Chronic Pain: A Primary Care Guide to Practical Management by Dawn A. Marcus

By Dawn A. Marcus

Sufferers with persistent discomfort current a different set of demanding situations to the first care clinician. In power ache: a first-rate Care consultant to useful administration, moment variation, top ache expert sunrise A. Marcus, MD, back deals useful, transparent, and succinct evidence-based methods to the prognosis and therapy of the myriad painful stipulations clinicians see of their workplaces each day, reminiscent of headache, again soreness, arthritis, fibromyalgia, and stomach ache. Sections addressing ache administration in young ones, pregnant girls, and seniors also are incorporated. This new version is designed to supply a practical method of assessing and treating the advanced concerns and features of persistent discomfort sufferers. New chapters extend upon the evidence-based thoughts and functional place of work instruments formerly supplied, with the addition of recent chapters addressing threat administration; soreness syndromes within the shoulder, higher extremity, and decrease extremity; and melanoma and end-of-life discomfort. persistent ache: a chief Care advisor to useful administration, moment version offers suggestions and methods which are designed to enhance the arrogance with which the first care health practitioner can strategy sufferers with complicated ache lawsuits, lessen employees pressure, and enhance sufferer good fortune.

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Extra resources for Chronic Pain: A Primary Care Guide to Practical Management (2nd Edition)

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Approach The serum glucose level is regulated by absorption, cellular uptake, gluconeogenesis, and glycogenolysis, which are regulated by the pancreas, intestines, liver, kidneys, and muscle. Hyperglycemia can result from perturbation of the hormones involved in glucose regulation, such as insulin and glucagon, and from dysfunction of the organs involved in glucose homeostasis. Diabetic ketoacidosis is decompensated catabolism triggered by a relative or absolute deficiency in insulin secretion.

Approach In the absence of renal failure, the fasting serum phosphate level is determined primarily according to the renal tubular reabsorption rate. A massive amount of phosphate can be released into the extracellular fluid via extensive cellular breakdown. Extensive rhabdomyolysis and hemolysis may cause hyperphosphatemia in the same way. Translocation of phosphate from cells in response to metabolic or respiratory alkalosis can lead to acute hyperphosphatemia. Chronic hyperphosphatemia is present in patients with hypoparathyroidism.

Patients with hypodipsia should receive a prescribed amount of water per day on a regular basis. 2 mg orally twice a day. • A low-salt diet along with use of thiazide diuretics that induce natriuresis is the treatment of choice for nephrogenic DI. Indomethacin has been used to treat drug-induced nephrogenic DI. , lithium) if clinically appropriate. 5 mEq/L) is perhaps the most common electrolyte abnormality in cancer patients. 5 mEq/L) usually are asymptomatic. In those with severe hypokalemia (less than 3 mEq/L), symptoms may range from mild to severe and are potentially fatal.

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