By Anil Bhansali, Anuradha Aggarwal, Girish Parthan, Yashpal Gogate
This e-book on pediatric endocrinology covers attention-grabbing and but frequently difficult instances between pediatric sufferers in a different Question-Answer structure. Simulating the bed-side case discussions through the ward rounds, one query logically ends up in one other, thereby producing interest and selling evidence-based drugs. Taking the readers throughout the complete spectrum ranging from etiology and pathophysiology to medical presentation to administration rules, each one query addresses one key point of the affliction. defined in an easy and lucid narrative, this booklet guarantees sound conceptual knowing whereas protecting every one subject comprehensively. This quantity covers very important subject matters reminiscent of brief stature, pubertal problems, thyroid issues, early life Cushing syndrome, rickets and osteomalacia, sickness of sexual differentiation and diabetes within the younger. much less universal illness akin to a number of endocrine neoplasia has additionally been integrated. those situations usually are not in basic terms noticeable by way of endocrinologists, yet also are controlled through paediatricians, internists, obstetricians and gynaecologists, orthopaedicians and surgeons.
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Additional info for Clinical Rounds in Endocrinology: Volume II - Pediatric Endocrinology
29. Which growth chart should be used for Indian children? The growth charts based on data from Indian population include Agarwal (1992, 1994), IAP (2007), Khadilkar (2009), Marwaha (2011), and revised IAP (2015) growth charts. Agarwal and Khadilkar charts were derived from school- going children belonging to affluent families, whereas Marwaha chart included school-going children from both upper and lower socioeconomic status. Data obtained from Agarwal et al. was used to derive IAP growth chart (2007), whereas data from nine studies in apparently healthy children from upper and middle socioeconomic class was used to derive revised IAP growth chart (2015).
What is pseudohypoparathyroidism? Pseudohypoparathyroidism is a metabolic bone disease characterised by typical features of Albright hereditary osteodystrophy including round facies, short stature, obesity and short metacarpals and metatarsals. Biochemiocally, high PTH with hypocalcemia and hyperphosphatemia are unique as opposed to idiopathic hypoparathyroidism which is characterised by low PTH. Further, basal ganglia calcification on plain CT helps in predicting the duration of the disease. The defect is confined to either PTH receptor or post-receptor signaling pathway.
13. What are the hormones responsible for GH-independent IGF1 generation? IGF1 generation is predominantly a GH-dependent phenomenon which is facilitated by thyroxine, insulin, and gonadal steroids. However, thyroxine, insulin, and gonadal steroids also promote GH-independent IGF1 generation. This is evidenced by the fact that children with GH deficiency/GH receptor mutation continues to grow, albeit at a lower height velocity, with measurable levels of serum IGF1, which suggest GH-independent IGF1 generation.